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In the context of tissue remodeling, GHK-Cu exerts dual-modulatory effects on the extracellular matrix (ECM)

MOTS-c is a mitochondrial-derived peptide that activates AMPK through direct mitochondrial signaling and nuclear translocation under metabolic stress, whereas metformin activates AMPK indirectly by inhibiting Complex I of the mitochondrial electron transport chain, and AICAR mimics AMP to trick the cell into AMPK activation

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It reduces pain and swelling significantly in the first weeks of the protocol